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N-Acetyl cysteine inhibits cell proliferation and differentiation of LPSInduced mc3t3-E1 cells via regulating inflammatory cytokines

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doi: 10.1186/s12974-018-1324-0 90 MohammadiM

Both overt T2DM and other insulin-resistant states are associated with chronic inflammation, enhanced reactive oxygen species generation, telomere shortening and cellular senescence processes that are driven in part by the obesity that commonly underlies T2DM [30]

The ability of a quinone to redox cycle is dependent upon the favourability of its reduction by a single electron, which can be quantitatively described by its reduction potential ( E o )

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